This browser is too old to run Achievable.

Update it, or open this page in a current version of Chrome, Safari, Firefox or Edge. On an iPhone or iPad, update iOS: every browser there runs on the version of Safari that comes with it.

Achievable logoAchievable logo
NPTE-PTA
Sign in
Sign up
Purchase
Textbook
Practice exams
Support
How it works
Resources
Exam catalog
Mountain with a flag at the peak
Textbook
Introduction
1. Cardiovascular system
2. Pulmonary system
3. Neuromuscular system
4. Pediatrics
5. Musculoskeletal system
5.1 Anatomy of musculoskeletal system
5.2 Anatomical terminology and exercise training principles
5.3 Joint mechanics and phases of healing
5.4 Upper extremity anatomy
5.5 Special tests of upper extremity
5.6 Comparing clinical presentation and interventions for upper extremity
5.7 Lower extremity anatomy
5.8 Special tests of lower extremity
5.9 Comparing clinical presentation and interventions for lower extremity
5.9.1 Hip and knee pathologies
5.9.2 Lower leg, ankle, and foot pathologies
5.10 Spine and pelvis anatomy
5.11 Special tests of the spine, pelvis, and temporomandibular joint
5.12 Comparing clinical presentation and interventions for the spine, pelvis, and temporomandibular joint
5.13 Other musculoskeletal conditions
5.14 Gait
5.15 Prosthetics
5.16 Orthotics
5.17 Medications, imaging, and fractures
5.18 Surgical protocols
6. Other systems
7. Non-systems
Wrapping up
Achievable logoAchievable logo
5.9.2 Lower leg, ankle, and foot pathologies
Achievable NPTE-PTA
5. Musculoskeletal system
5.9. Comparing clinical presentation and interventions for lower extremity
Our NPTE-PTA course is now in "early access" - get 50% off for a limited time.

Lower leg, ankle, and foot pathologies

9 min read
Font
Discuss
Share
Feedback

Acetaminophen and NSAIDs are the standard first-line analgesic/anti-inflammatory management for the conditions on this page; additional or different medical management is called out where it applies.

Conditions of the lower leg

Acute compartment syndrome

  • Increased compartment pressure of the lower leg (specifically, the anterior, lateral, and posterior areas)

    • Commonly caused by direct trauma or fracture
  • Symptoms (six P’s)

    • Pain in the lower leg, out of proportion to the injury and worse with passive stretch of the compartment muscles
    • Poikilothermia (coolness) in the lower leg
    • Paresthesia to the lower leg - specifically to the deep peroneal nerve
    • Paralysis/paresis of the lower leg, specifically of the deep peroneal nerve
    • Pallor to the lower leg
    • Pulselessness in the dorsal pedal artery

Pain and paresthesia are the earliest signs to appear. A palpable pulse does not rule out compartment syndrome - pulselessness is a late sign.

  • Diagnosis
    • Clinical presentation
  • Medical management
    • Medical emergency: immediate fasciotomy is indicated to relieve pressure and decrease the likelihood of prolonged neurovascular compromise
  • Physical therapy management
    • Post-operatively, physical therapy to ensure return to normal function

PTA role and pitfall: Acute compartment syndrome is a medical emergency, not a condition to treat with exercise. If any of the six P’s emerge or worsen during a session, stop the activity, take vital/neurovascular measurements, and notify the supervising PT or physician immediately rather than continuing the plan of care. This is different from chronic exertional compartment syndrome (below), which develops gradually with activity and resolves with rest.

Cross-sectional view of lower leg compartments and bones.
Cross-sectional view of lower leg
Achievable

Chronic exertional compartment syndrome

  • Increased compartment pressure of the lower leg (specifically, the anterior, lateral, and posterior areas), causing a restriction in blood flow

    • Develops gradually over a period of time due to repetitive movements and exertional activities
    • Most often involves the anterior compartment and is frequently bilateral
  • Symptoms

    • Pain
    • Numbness and tingling
    • Weakness
    • Swelling
    • Tightness
    • Foot drop
  • Diagnosis

    • Clinical presentation
    • Compartment pressure testing before and after exercise
  • Medical management

    • Fasciotomy if symptoms persist despite activity modification
  • Physical therapy management

    • Functional training, proprioceptive training
    • Decrease exacerbating activity
    • May require the use of orthoses

Stress fractures

  • Microfractures to the tibia, fibula, or bones of the foot and ankle due to overuse (repetitive micro-traumas to the tibia or fibula)

    • Commonly due to poor alignment of the lower extremity when performing activity, deconditioning, or improper training
  • Symptoms

    • Localized sharp pain that gradually worsens with activity
    • Mild swelling may occur around the fracture site
    • The affected area may feel weak or unstable
    • Difficult to put weight on the injured area
    • Early pain eases with rest; pain at rest develops as the injury progresses
  • Diagnosis

    • Clinical presentation reproduced with percussion
    • X-rays are often negative for the first 2-3 weeks; bone scan or MRI confirms the diagnosis
  • Physical therapy management

    • Initially decreased exacerbating activity
    • Correction of malalignment and biomechanical issues
    • Strengthening and coordinating activities

Medial tibial stress syndrome (shin splints) causes similar activity-related pain but is diffuse along the tibia without a focal point, unlike the localized point tenderness of a stress fracture. Treatment of shin splints begins with several weeks of rest from the activity that caused the pain and cold packs for 20 minutes at a time, several times a day.

Ankle and foot conditions

Ligament sprains

  • Classification of sprains

    • Grade 1
      • A mild sprain where the ligament is stretched or slightly torn
      • Symptoms include minor swelling, tenderness, and bruising
      • Recovery time is usually 1-3 weeks
    • Grade 2
      • A moderate sprain where the ligament is partially torn
      • Symptoms include swelling, bruising, pain, and difficulty walking
      • Recovery time is usually 3-6 weeks
    • Grade 3
      • A severe sprain where the ligament is completely torn
      • Symptoms include severe pain, swelling, bruising, and instability
      • Recovery time can be several months
  • A high ankle sprain injures the ligaments between the tibia and fibula when the foot is flexed upward and then twisted; squeezing the tibia and fibula together causes pain higher up the leg

  • Symptoms

    • See specific grade for symptoms associated
  • Diagnosis

    • Clinical presentation, ligament laxity
      • Special tests:
        • Anterior drawer (Anterior talofibular ligament)
        • Talar tilt (inversion stresses the calcaneofibular ligament; eversion stresses the deltoid ligament)
  • Medical management

    • Grade 2: immobilization device may be recommended
    • Grade 3 sprains are usually managed conservatively with immobilization/bracing and functional rehabilitation; surgery is reserved for chronic instability or failed conservative care
  • Physical therapy management

    • Grade 1: conservative management of rest, ice, compression, and elevation acutely; then functional mobility training
    • Grades 2 and 3:
      • Functional mobility training
      • Biomechanical fault realignment
      • Joint protection and mobility
      • Post-surgical protocols if surgery is indicated

Achilles tendinopathy

  • Chronic degeneration (tendinosis) of the Achilles tendon due to overuse, age, rheumatoid arthritis, or gout

  • Symptoms

    • Pain, typically located in the back of the heel or just above it
    • Tenderness and stiffness in the Achilles tendon
    • Swelling and redness around the tendon
    • Pain that worsens with activity and improves with rest
  • Diagnosis

    • Clinical presentation
      • Thompson’s test to rule out Achilles tendon rupture
  • Medical management

    • Corticosteroid injection is generally avoided (risk of tendon weakening and rupture)
    • Management of rheumatoid arthritis or gout
  • Physical therapy management

    • Interventions follow the PT’s plan of care for the stage of healing (for example, relative rest and pain control acutely, then progressive tendon loading - eccentric or heavy slow resistance calf exercise, the first-line evidence-based intervention)
    • The PTA monitors the pain response to loading and reports any worsening, or a sudden pop or loss of push-off strength, to the supervising PT

Peroneal tendon subluxation

  • Peroneal tendon subluxation is linked to laxity or rupture of the superior peroneal retinaculum and causes painful clicking or popping at the lateral malleolus

Plantar fasciitis

  • Degeneration or irritation of the plantar fascia at its origin on the medial calcaneal tubercle, caused by overuse, tight calf musculature, or biomechanical faults such as pes planus or pes cavus

  • Symptoms

    • Classic first-step pain: sharp heel pain with the first steps in the morning or after rest, which eases somewhat with continued activity
    • Tenderness at the medial calcaneal tubercle
  • Diagnosis

    • Clinical presentation and palpation at the medial calcaneal tubercle
  • Medical management

    • NSAIDs; corticosteroid injection only for refractory cases (risk of fascia rupture or fat pad atrophy)
  • Physical therapy management

    • Plantar fascia and calf stretching
    • Taping or orthoses
    • Night splints

Plantar fasciitis and tarsal tunnel syndrome both cause plantar foot pain but present differently: plantar fasciitis produces localized first-step heel pain at the medial calcaneal tubercle, while tarsal tunnel syndrome produces neural burning or tingling with a positive Tinel’s sign.

Tarsal tunnel syndrome

  • Entrapment of the posterior tibial nerve within the tarsal tunnel

    • Caused by compression from overpronation/flat foot, trauma, space-occupying lesions (ganglion, varicosities), or tenosynovitis of the medial ankle tendons
  • Symptoms

    • Numbness and tingling in the sole, arch, or toes.
    • Burning or shooting pain may radiate from the ankle to the foot or calf
    • Swelling or redness
    • Pain that improves with rest
  • Diagnosis

    • Clinical presentation
      • Special test: Tinel’s sign
    • Electrodiagnostic testing
  • Medical management

    • Surgical intervention if conservative management is not effective
  • Physical therapy management

    • Joint mobility
    • Functional exercise training
    • Orthoses
    • Neurotension stretching

Posterior tibial tendon dysfunction

  • Posterior tibial tendon dysfunction is the most common cause of adult-acquired flatfoot; it begins with medial ankle and foot pain, and collapse of the medial longitudinal arch follows

Foot deformities

  • Pes cavus - high arch foot

    • Due to genetics or biomechanical faults
  • Pes planus - absent arch foot, overpronation

    • Due to genetics, age, obesity, or biomechanical faults
  • Equinus - plantar flexed foot

    • Can be due to genetics, bone deformity, or neurological conditions
  • Talipes equinovarus - club foot (plantar flexion and inversion)

    • Due to congenital deformity
  • Rearfoot varus - inversion of the calcaneus when the subtalar joint is in neutral

  • Rearfoot valgus - eversion of the calcaneus when the subtalar joint is in neutral

  • Forefoot varus - inversion of the forefoot when the subtalar joint is in neutral

  • Forefoot valgus - eversion of the forefoot when the subtalar joint is in neutral

  • Symptoms

    • Observation of the foot as noted above; abnormalities in gait due to uneven levers
  • Diagnosis

    • Clinical presentation and observation
  • Medical management

    • Management of underlying causes as appropriate; congenital clubfoot (talipes equinovarus) is corrected with serial stretching and casting (Ponseti method) starting shortly after birth
  • Physical therapy management

    • Improved biomechanical alignment
    • Orthoses as appropriate
    • Joint mobility
    • Patient education

Tarsal coalition

  • A tarsal coalition is an abnormal connection of two or more foot bones that can cause a severe, rigid flatfoot, with symptoms usually appearing in late childhood or early adolescence as the connection hardens

Charcot-Marie-Tooth disease

  • Progressive disorder causing peroneal muscular atrophy and involvement of the peroneal nerves - causing motor and sensory deficits

    • Genetic disorder
    • Begins in the lower legs but can progress to the forearm and hands
  • Symptoms

    • Foot and leg weakness
    • Foot abnormalities: high arches, hammertoes, and flat feet
    • Foot drop with a steppage gait due to weakness of the ankle dorsiflexors
    • Numbness, tingling, and burning sensations in the lower leg
    • Muscle atrophy in the feet and legs
    • Clumsiness and difficulty with fine motor skills
  • Diagnosis

    • Clinical presentation
    • Electrodiagnostic exam
    • Genetic testing
  • Medical management

    • Surgical correction of joint abnormalities as deemed appropriate
    • Chronic inflammatory demyelinating polyneuropathy (CIDP), an acquired peripheral neuropathy, is treated with intravenous immune globulin and glucocorticoids
  • Physical therapy management

    • Progressive disorder with no cure, so physical therapy follows the PT’s plan of care and centers on patient education, skin assessments for possible development of wounds, contracture prevention/management, and functional mobility training

Compartment syndromes

  • Acute compartment syndrome: medical emergency from trauma/fracture; six P’s (pain out of proportion/with passive stretch, poikilothermia, paresthesia, paralysis, pallor, pulselessness)
    • Pain and paresthesia appear earliest; pulselessness is a late sign; palpable pulse does NOT rule it out
    • Requires immediate fasciotomy; PTA must stop activity and alert PT/physician if any P’s emerge
  • Chronic exertional compartment syndrome: gradual onset with repetitive activity, usually anterior compartment, often bilateral
    • Diagnosed via pre/post-exercise compartment pressure testing
    • Managed with activity modification, functional/proprioceptive training, orthoses; fasciotomy only if conservative care fails

Stress fractures vs. shin splints

  • Stress fractures: overuse microfractures, localized sharp pain worsening with activity, point tenderness with percussion
    • X-rays often negative early (2-3 weeks); bone scan/MRI confirms
    • PT: reduce aggravating activity, correct malalignment, strengthening
  • Medial tibial stress syndrome (shin splints): diffuse tibial pain, no focal point (contrast with stress fracture’s localized tenderness)

Ligament sprains

  • Graded 1 (mild stretch/slight tear) to 3 (complete tear) based on severity, symptoms, and recovery time (1-3 wks to several months)
  • Special tests: anterior drawer (ATFL), talar tilt (inversion=calcaneofibular ligament, eversion=deltoid ligament)
  • Grade 2 may need immobilization; Grade 3 usually conservative (bracing) unless chronic instability
  • PT: Grade 1 RICE acutely, then functional mobility; Grades 2-3 functional/biomechanical rehab, joint protection, post-op protocols if surgical

Achilles tendinopathy & plantar fasciitis

  • Achilles tendinopathy: chronic degeneration from overuse/age/RA/gout; pain worsens with activity, improves with rest
    • Thompson’s test rules out rupture; avoid corticosteroid injections (rupture risk)
    • PT: progressive tendon loading (eccentric/heavy slow resistance) is first-line; monitor for pop/loss of push-off strength
  • Plantar fasciitis: classic first-step morning heel pain, tenderness at medial calcaneal tubercle
    • PT: fascia/calf stretching, taping/orthoses, night splints; corticosteroids only for refractory cases

Tarsal tunnel syndrome

  • Posterior tibial nerve entrapment from overpronation, trauma, lesions, or tenosynovitis
  • Presents with neural burning/tingling/numbness in sole/arch/toes, positive Tinel’s sign (contrasts with localized plantar fasciitis pain)
  • PT: joint mobility, orthoses, neurotension stretching; surgery if conservative care fails

Foot deformities & Charcot-Marie-Tooth

  • Deformities classified by arch (pes cavus/planus), ankle position (equinus, clubfoot), and forefoot/rearfoot varus/valgus alignment relative to subtalar neutral
  • Clubfoot corrected via Ponseti method (serial casting/stretching) soon after birth
  • Charcot-Marie-Tooth: genetic, progressive peroneal nerve/muscle disorder causing foot drop, steppage gait, high arches/hammertoes, sensory loss
    • No cure; PT focuses on education, skin checks, contracture prevention, functional mobility

Sign up for free to take 21 quiz questions on this topic

Previous
Next  | 5.10 Spine and pelvis anatomy
All rights reserved ©2016 - 2026 Achievable, Inc.

Lower leg, ankle, and foot pathologies

Acetaminophen and NSAIDs are the standard first-line analgesic/anti-inflammatory management for the conditions on this page; additional or different medical management is called out where it applies.

Conditions of the lower leg

Acute compartment syndrome

  • Increased compartment pressure of the lower leg (specifically, the anterior, lateral, and posterior areas)

    • Commonly caused by direct trauma or fracture
  • Symptoms (six P’s)

    • Pain in the lower leg, out of proportion to the injury and worse with passive stretch of the compartment muscles
    • Poikilothermia (coolness) in the lower leg
    • Paresthesia to the lower leg - specifically to the deep peroneal nerve
    • Paralysis/paresis of the lower leg, specifically of the deep peroneal nerve
    • Pallor to the lower leg
    • Pulselessness in the dorsal pedal artery

Pain and paresthesia are the earliest signs to appear. A palpable pulse does not rule out compartment syndrome - pulselessness is a late sign.

  • Diagnosis
    • Clinical presentation
  • Medical management
    • Medical emergency: immediate fasciotomy is indicated to relieve pressure and decrease the likelihood of prolonged neurovascular compromise
  • Physical therapy management
    • Post-operatively, physical therapy to ensure return to normal function

PTA role and pitfall: Acute compartment syndrome is a medical emergency, not a condition to treat with exercise. If any of the six P’s emerge or worsen during a session, stop the activity, take vital/neurovascular measurements, and notify the supervising PT or physician immediately rather than continuing the plan of care. This is different from chronic exertional compartment syndrome (below), which develops gradually with activity and resolves with rest.

Chronic exertional compartment syndrome

  • Increased compartment pressure of the lower leg (specifically, the anterior, lateral, and posterior areas), causing a restriction in blood flow

    • Develops gradually over a period of time due to repetitive movements and exertional activities
    • Most often involves the anterior compartment and is frequently bilateral
  • Symptoms

    • Pain
    • Numbness and tingling
    • Weakness
    • Swelling
    • Tightness
    • Foot drop
  • Diagnosis

    • Clinical presentation
    • Compartment pressure testing before and after exercise
  • Medical management

    • Fasciotomy if symptoms persist despite activity modification
  • Physical therapy management

    • Functional training, proprioceptive training
    • Decrease exacerbating activity
    • May require the use of orthoses

Stress fractures

  • Microfractures to the tibia, fibula, or bones of the foot and ankle due to overuse (repetitive micro-traumas to the tibia or fibula)

    • Commonly due to poor alignment of the lower extremity when performing activity, deconditioning, or improper training
  • Symptoms

    • Localized sharp pain that gradually worsens with activity
    • Mild swelling may occur around the fracture site
    • The affected area may feel weak or unstable
    • Difficult to put weight on the injured area
    • Early pain eases with rest; pain at rest develops as the injury progresses
  • Diagnosis

    • Clinical presentation reproduced with percussion
    • X-rays are often negative for the first 2-3 weeks; bone scan or MRI confirms the diagnosis
  • Physical therapy management

    • Initially decreased exacerbating activity
    • Correction of malalignment and biomechanical issues
    • Strengthening and coordinating activities

Medial tibial stress syndrome (shin splints) causes similar activity-related pain but is diffuse along the tibia without a focal point, unlike the localized point tenderness of a stress fracture. Treatment of shin splints begins with several weeks of rest from the activity that caused the pain and cold packs for 20 minutes at a time, several times a day.

Ankle and foot conditions

Ligament sprains

  • Classification of sprains

    • Grade 1
      • A mild sprain where the ligament is stretched or slightly torn
      • Symptoms include minor swelling, tenderness, and bruising
      • Recovery time is usually 1-3 weeks
    • Grade 2
      • A moderate sprain where the ligament is partially torn
      • Symptoms include swelling, bruising, pain, and difficulty walking
      • Recovery time is usually 3-6 weeks
    • Grade 3
      • A severe sprain where the ligament is completely torn
      • Symptoms include severe pain, swelling, bruising, and instability
      • Recovery time can be several months
  • A high ankle sprain injures the ligaments between the tibia and fibula when the foot is flexed upward and then twisted; squeezing the tibia and fibula together causes pain higher up the leg

  • Symptoms

    • See specific grade for symptoms associated
  • Diagnosis

    • Clinical presentation, ligament laxity
      • Special tests:
        • Anterior drawer (Anterior talofibular ligament)
        • Talar tilt (inversion stresses the calcaneofibular ligament; eversion stresses the deltoid ligament)
  • Medical management

    • Grade 2: immobilization device may be recommended
    • Grade 3 sprains are usually managed conservatively with immobilization/bracing and functional rehabilitation; surgery is reserved for chronic instability or failed conservative care
  • Physical therapy management

    • Grade 1: conservative management of rest, ice, compression, and elevation acutely; then functional mobility training
    • Grades 2 and 3:
      • Functional mobility training
      • Biomechanical fault realignment
      • Joint protection and mobility
      • Post-surgical protocols if surgery is indicated

Achilles tendinopathy

  • Chronic degeneration (tendinosis) of the Achilles tendon due to overuse, age, rheumatoid arthritis, or gout

  • Symptoms

    • Pain, typically located in the back of the heel or just above it
    • Tenderness and stiffness in the Achilles tendon
    • Swelling and redness around the tendon
    • Pain that worsens with activity and improves with rest
  • Diagnosis

    • Clinical presentation
      • Thompson’s test to rule out Achilles tendon rupture
  • Medical management

    • Corticosteroid injection is generally avoided (risk of tendon weakening and rupture)
    • Management of rheumatoid arthritis or gout
  • Physical therapy management

    • Interventions follow the PT’s plan of care for the stage of healing (for example, relative rest and pain control acutely, then progressive tendon loading - eccentric or heavy slow resistance calf exercise, the first-line evidence-based intervention)
    • The PTA monitors the pain response to loading and reports any worsening, or a sudden pop or loss of push-off strength, to the supervising PT

Peroneal tendon subluxation

  • Peroneal tendon subluxation is linked to laxity or rupture of the superior peroneal retinaculum and causes painful clicking or popping at the lateral malleolus

Plantar fasciitis

  • Degeneration or irritation of the plantar fascia at its origin on the medial calcaneal tubercle, caused by overuse, tight calf musculature, or biomechanical faults such as pes planus or pes cavus

  • Symptoms

    • Classic first-step pain: sharp heel pain with the first steps in the morning or after rest, which eases somewhat with continued activity
    • Tenderness at the medial calcaneal tubercle
  • Diagnosis

    • Clinical presentation and palpation at the medial calcaneal tubercle
  • Medical management

    • NSAIDs; corticosteroid injection only for refractory cases (risk of fascia rupture or fat pad atrophy)
  • Physical therapy management

    • Plantar fascia and calf stretching
    • Taping or orthoses
    • Night splints

Plantar fasciitis and tarsal tunnel syndrome both cause plantar foot pain but present differently: plantar fasciitis produces localized first-step heel pain at the medial calcaneal tubercle, while tarsal tunnel syndrome produces neural burning or tingling with a positive Tinel’s sign.

Tarsal tunnel syndrome

  • Entrapment of the posterior tibial nerve within the tarsal tunnel

    • Caused by compression from overpronation/flat foot, trauma, space-occupying lesions (ganglion, varicosities), or tenosynovitis of the medial ankle tendons
  • Symptoms

    • Numbness and tingling in the sole, arch, or toes.
    • Burning or shooting pain may radiate from the ankle to the foot or calf
    • Swelling or redness
    • Pain that improves with rest
  • Diagnosis

    • Clinical presentation
      • Special test: Tinel’s sign
    • Electrodiagnostic testing
  • Medical management

    • Surgical intervention if conservative management is not effective
  • Physical therapy management

    • Joint mobility
    • Functional exercise training
    • Orthoses
    • Neurotension stretching

Posterior tibial tendon dysfunction

  • Posterior tibial tendon dysfunction is the most common cause of adult-acquired flatfoot; it begins with medial ankle and foot pain, and collapse of the medial longitudinal arch follows

Foot deformities

  • Pes cavus - high arch foot

    • Due to genetics or biomechanical faults
  • Pes planus - absent arch foot, overpronation

    • Due to genetics, age, obesity, or biomechanical faults
  • Equinus - plantar flexed foot

    • Can be due to genetics, bone deformity, or neurological conditions
  • Talipes equinovarus - club foot (plantar flexion and inversion)

    • Due to congenital deformity
  • Rearfoot varus - inversion of the calcaneus when the subtalar joint is in neutral

  • Rearfoot valgus - eversion of the calcaneus when the subtalar joint is in neutral

  • Forefoot varus - inversion of the forefoot when the subtalar joint is in neutral

  • Forefoot valgus - eversion of the forefoot when the subtalar joint is in neutral

  • Symptoms

    • Observation of the foot as noted above; abnormalities in gait due to uneven levers
  • Diagnosis

    • Clinical presentation and observation
  • Medical management

    • Management of underlying causes as appropriate; congenital clubfoot (talipes equinovarus) is corrected with serial stretching and casting (Ponseti method) starting shortly after birth
  • Physical therapy management

    • Improved biomechanical alignment
    • Orthoses as appropriate
    • Joint mobility
    • Patient education

Tarsal coalition

  • A tarsal coalition is an abnormal connection of two or more foot bones that can cause a severe, rigid flatfoot, with symptoms usually appearing in late childhood or early adolescence as the connection hardens

Charcot-Marie-Tooth disease

  • Progressive disorder causing peroneal muscular atrophy and involvement of the peroneal nerves - causing motor and sensory deficits

    • Genetic disorder
    • Begins in the lower legs but can progress to the forearm and hands
  • Symptoms

    • Foot and leg weakness
    • Foot abnormalities: high arches, hammertoes, and flat feet
    • Foot drop with a steppage gait due to weakness of the ankle dorsiflexors
    • Numbness, tingling, and burning sensations in the lower leg
    • Muscle atrophy in the feet and legs
    • Clumsiness and difficulty with fine motor skills
  • Diagnosis

    • Clinical presentation
    • Electrodiagnostic exam
    • Genetic testing
  • Medical management

    • Surgical correction of joint abnormalities as deemed appropriate
    • Chronic inflammatory demyelinating polyneuropathy (CIDP), an acquired peripheral neuropathy, is treated with intravenous immune globulin and glucocorticoids
  • Physical therapy management

    • Progressive disorder with no cure, so physical therapy follows the PT’s plan of care and centers on patient education, skin assessments for possible development of wounds, contracture prevention/management, and functional mobility training
Key points

Compartment syndromes

  • Acute compartment syndrome: medical emergency from trauma/fracture; six P’s (pain out of proportion/with passive stretch, poikilothermia, paresthesia, paralysis, pallor, pulselessness)
    • Pain and paresthesia appear earliest; pulselessness is a late sign; palpable pulse does NOT rule it out
    • Requires immediate fasciotomy; PTA must stop activity and alert PT/physician if any P’s emerge
  • Chronic exertional compartment syndrome: gradual onset with repetitive activity, usually anterior compartment, often bilateral
    • Diagnosed via pre/post-exercise compartment pressure testing
    • Managed with activity modification, functional/proprioceptive training, orthoses; fasciotomy only if conservative care fails

Stress fractures vs. shin splints

  • Stress fractures: overuse microfractures, localized sharp pain worsening with activity, point tenderness with percussion
    • X-rays often negative early (2-3 weeks); bone scan/MRI confirms
    • PT: reduce aggravating activity, correct malalignment, strengthening
  • Medial tibial stress syndrome (shin splints): diffuse tibial pain, no focal point (contrast with stress fracture’s localized tenderness)

Ligament sprains

  • Graded 1 (mild stretch/slight tear) to 3 (complete tear) based on severity, symptoms, and recovery time (1-3 wks to several months)
  • Special tests: anterior drawer (ATFL), talar tilt (inversion=calcaneofibular ligament, eversion=deltoid ligament)
  • Grade 2 may need immobilization; Grade 3 usually conservative (bracing) unless chronic instability
  • PT: Grade 1 RICE acutely, then functional mobility; Grades 2-3 functional/biomechanical rehab, joint protection, post-op protocols if surgical

Achilles tendinopathy & plantar fasciitis

  • Achilles tendinopathy: chronic degeneration from overuse/age/RA/gout; pain worsens with activity, improves with rest
    • Thompson’s test rules out rupture; avoid corticosteroid injections (rupture risk)
    • PT: progressive tendon loading (eccentric/heavy slow resistance) is first-line; monitor for pop/loss of push-off strength
  • Plantar fasciitis: classic first-step morning heel pain, tenderness at medial calcaneal tubercle
    • PT: fascia/calf stretching, taping/orthoses, night splints; corticosteroids only for refractory cases

Tarsal tunnel syndrome

  • Posterior tibial nerve entrapment from overpronation, trauma, lesions, or tenosynovitis
  • Presents with neural burning/tingling/numbness in sole/arch/toes, positive Tinel’s sign (contrasts with localized plantar fasciitis pain)
  • PT: joint mobility, orthoses, neurotension stretching; surgery if conservative care fails

Foot deformities & Charcot-Marie-Tooth

  • Deformities classified by arch (pes cavus/planus), ankle position (equinus, clubfoot), and forefoot/rearfoot varus/valgus alignment relative to subtalar neutral
  • Clubfoot corrected via Ponseti method (serial casting/stretching) soon after birth
  • Charcot-Marie-Tooth: genetic, progressive peroneal nerve/muscle disorder causing foot drop, steppage gait, high arches/hammertoes, sensory loss
    • No cure; PT focuses on education, skin checks, contracture prevention, functional mobility

More from Comparing clinical presentation and interventions for lower extremity

  • Hip and knee pathologies