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Introduction
1. Cardiovascular system
2. Pulmonary system
3. Neuromuscular system
4. Pediatrics
5. Musculoskeletal system
6. Other systems
6.1 Integumentary system
6.2 Lymphedema
6.3 Gastrointestinal and reproductive systems
6.4 Urinary incontinence and renal disorders
6.5 Endocrine and metabolic systems
6.6 Cancer and psychological conditions
6.7 Infectious disease, immune disorders, and hematological disorders
7. Non-systems
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6.4 Urinary incontinence and renal disorders
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6. Other systems
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Urinary incontinence and renal disorders

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Incontinence

Urinary incontinence is the involuntary loss of urine due to dysfunction in the bladder, urethra, or pelvic floor muscles. There are several types, each with different causes and treatments; urodynamic studies evaluate bladder function to classify the type.

Stress incontinence

Cause

  • Weak pelvic floor muscles or urethral sphincter dysfunction
  • Common in postpartum women, postmenopausal women, and after prostate surgery in men
  • Triggered by increased intra-abdominal pressure (e.g., coughing, sneezing, laughing, jumping)

Treatment

  • Pelvic floor muscle training (Kegels) to strengthen muscles: cue a lift-and-squeeze contraction around the urethra and anus without substituting gluteal, hip-adductor, or abdominal muscles. Training progresses from brief, quick contractions (useful for urge suppression) to longer-held endurance contractions, and a bladder diary (tracking fluid intake, voiding times, and leakage episodes) helps guide the plan.
  • Bladder training to improve control
  • Biofeedback & electrical stimulation for muscle activation
  • Pessary (vaginal) or urethral inserts (in some cases to support and hold pelvic organs in place)
  • Surgical options (e.g., sling procedures) for severe cases

Urge incontinence

Cause

  • Detrusor muscle overactivity leads to sudden, strong urges to urinate
  • Can be related to neurological conditions (e.g., stroke, Parkinson’s, MS), infections, or idiopathic causes
    • In older adults a common infection, such as a urinary tract infection, pneumonia or influenza, can cause delirium, which begins suddenly, whereas dementia typically begins gradually

Treatment

  • Bladder retraining (scheduled voiding, delaying urination)
  • Pelvic floor exercises to control urgency
  • Behavioral modifications (reducing caffeine, alcohol, and bladder irritants)
  • Medications (anticholinergics, beta-3 agonists) to relax the bladder
  • Neuromodulation (e.g., tibial nerve stimulation) in severe cases

Overflow incontinence

Cause

  • Incomplete bladder emptying due to obstruction (e.g., enlarged prostate, pelvic organ prolapse) or weak detrusor muscle (e.g., diabetes, spinal cord injury, multiple sclerosis)
  • Symptoms include dribbling, a weak stream, and a feeling of incomplete emptying
  • In spinal cord injury at T6 or above, a distended bladder or a blocked catheter is the most common trigger of autonomic dysreflexia (sudden hypertension, pounding headache, flushing above the lesion): sit the patient up, check the catheter, and notify the PT immediately

Treatment

  • Double voiding technique (waiting and trying to urinate again)
  • Timed voiding to prevent overflow
  • Pelvic floor strengthening does not treat overflow incontinence, because it neither relieves an obstruction nor strengthens a weak detrusor; if a pelvic floor that fails to relax is impeding emptying, pelvic floor relaxation training (often with biofeedback) is used instead
  • Catheterization (intermittent or long-term) for severe cases
  • Surgical intervention if caused by obstruction (e.g., prostate surgery)

Functional incontinence

Cause

  • Physical, structural, or cognitive impairments (e.g., stroke, dementia, arthritis, Parkinson’s, steps to bathroom, physical obstacles) prevent timely access to the bathroom
  • No direct bladder dysfunction, but mobility limitations or mental impairments lead to accidents

Treatment

  • Environmental modifications (accessible bathrooms, commodes, grab bars)
  • Scheduled toileting based on routine
  • Mobility training & assistive devices
  • Caregiver education for dementia-related cases

The following table compares the four types of urinary incontinence by cause, presentation, and first-line physical therapy management.


Type Cause Presentation First-line PT management
Stress Weak pelvic floor muscles or urethral sphincter Leakage with coughing, sneezing, laughing, or jumping Pelvic floor muscle training (Kegels), biofeedback
Urge Detrusor overactivity Sudden, strong urge; leakage before reaching the toilet Bladder retraining (scheduled voiding, delaying urination), behavioral modification
Overflow Obstruction or a weak detrusor, so the bladder does not empty Dribbling, weak stream, feeling of incomplete emptying Double and timed voiding, catheterization when needed; not pelvic floor strengthening
Functional Mobility or cognitive barriers; the bladder itself works Accidents because the toilet cannot be reached in time Environmental modification, scheduled toileting, mobility training

Pelvic floor function and exercise precautions

A correct pelvic floor contraction squeezes around the pelvic openings and lifts the pelvic floor; the perineum moves inward and up, not outward. Two everyday uses follow from that:

  • The knack: contracting the pelvic floor just before a cough, sneeze, or lift, so it is already braced when abdominal pressure rises.
  • Normal voiding: about 6-8 voids a day, every 3-4 hours. A bladder diary is read against this interval, and timed voiding works toward it.
  • Nocturia: the need to get up regularly at night to urinate; about 50% of adults older than 65 get up at least once a night, and about a quarter have two or more episodes.

Bladder and pelvic floor programs have their own adverse effects to watch for and report to the PT:

  • Bearing down instead of lifting: straining (pushing downward, often with breath-holding) is a common error that raises intra-abdominal pressure and loads the pelvic floor - the opposite of the exercise’s goal.
  • Overactive pelvic floor: a pelvic floor that can’t fully relax is linked to pelvic pain, urinary frequency and urgency, and difficulty with bowel movements. Strengthening isn’t always appropriate here; relaxation training is used instead.
  • Fluid restriction: patients often drink less to leak less, but concentrated urine irritates the bladder and promotes urgency, frequency, and urinary tract infections, and restricting fluids worsens constipation.

Interventions for other problems can also provoke leakage:

  • High-impact and heavy-lift exercise: running, jumping, landing, and heavy lifting raise intra-abdominal pressure, and incontinence is common in high-impact sports and in weightlifters. Report new leakage during exercise to the PT.
  • Diuretics: increase urine production, causing frequency and urgency, and can lead to urge leakage in an older patient who moves slowly. Plan bathroom access, and where possible schedule sessions away from the hours right after the dose.

Renal disorders

Common renal disorders

The kidneys play a vital role in filtering waste, regulating electrolytes, and maintaining fluid balance. Dysfunction can lead to serious systemic issues, including metabolic imbalances and cardiovascular complications.

Acute kidney injury (AKI)

Definition: Sudden loss of kidney function due to ischemia, toxins, or trauma. It is often reversible if treated early.

Causes:

  • Pre-renal: Hypovolemia, shock, heart failure.
  • Intra-renal: Acute tubular necrosis, nephrotoxic drugs, infections
  • Post-renal: Obstruction (kidney stones, tumors, BPH). Kidney stones (renal calculi) typically present with sudden-onset flank pain and hematuria; as the stone travels down the ureter, pain can radiate to the lower abdomen, groin, and genital region. This radiating pain can mimic musculoskeletal pain, so report it to the supervising PT rather than treating it as a musculoskeletal complaint.

Signs and symptoms:

  • Oliguria (low urine output) and fluid retention
  • Electrolyte imbalance (increased potassium) and abnormal labs (increased BUN and creatinine)

PT considerations:

  • Monitor for fatigue, altered mental status, and electrolyte imbalances
  • Avoid overexertion due to risk of hypotension and electrolyte shifts

Chronic kidney disease (CKD)

Definition: Progressive decline in kidney function, leading to end-stage renal disease (ESRD) if untreated

Causes:

  • Diabetes (leading cause), hypertension, glomerulonephritis, polycystic kidney disease

The following table lists the five stages of chronic kidney disease by glomerular filtration rate (GFR) and clinical picture.


Stage GFR (mL/min/1.73 m²) Clinical picture
1 ≥90 (with kidney damage) Mild damage, no major symptoms
2 60-89 Mild damage, no major symptoms
3 30-59 Moderate impairment; possible anemia, bone mineral disease
4 15-29 Severe dysfunction; dialysis preparation begins
5 (ESRD) <15, or on dialysis Requires dialysis or a kidney transplant

Approximate normal reference ranges are serum creatinine 0.6-1.2 mg/dL, BUN 7-20 mg/dL, and potassium 3.5-5.0 mEq/L; values above these ranges reflect the waste-product and electrolyte imbalances described below. Imaging: renal ultrasound checks for obstruction, stones, or structural changes.

Signs and symptoms:

  • Fatigue, muscle cramps, edema, anemia, metabolic acidosis, hypertension, pruritus (intense itching, most common in advanced CKD and ESRD)
  • Uremia (buildup of waste in blood) → nausea, confusion, neuropathy

PT considerations:

  • Monitor for fatigue, blood pressure changes, and electrolyte imbalances
  • Encourage low-to-moderate intensity exercise to improve functional capacity
  • Avoid excessive fluid loss (dehydration can worsen kidney function)
    • Dehydration causes dry mouth and reduced urine production, and in severe cases blood pressure falls, causing light-headedness or fainting, particularly upon standing
  • Be aware of bone mineral disease & risk of fractures

Nephrotic syndrome

Definition: A kidney disorder causing severe protein loss in urine due to damage to the glomeruli

Signs and symptoms:

  • Severe edema (especially in the legs), proteinuria (>3.5g/day), hypoalbuminemia, hyperlipidemia.
  • Increased risk of thrombosis and infections

PT considerations:

  • Monitor for edema-related mobility restrictions
  • Be cautious of increased clotting risk during exercise

Hemodialysis (HD) considerations

Hemodialysis is used in ESRD to filter blood through a machine when the kidneys can no longer perform their function. It is typically done 3 times per week for 3-5 hours per session.

Common complications:

  • Hypotension during or after dialysis (due to rapid fluid removal)
  • Fatigue, dizziness, cramping (electrolyte shifts)
  • Access site issues (fistula/graft infections, thrombosis)

PT considerations:

  • Schedule exercise on non-dialysis days or at least 4 hours after treatment; avoid exercise immediately before or after dialysis because of fatigue and hypotension risk
  • Do not take blood pressure on the arm with an AV fistula/graft to prevent complications
  • Encourage low-impact aerobic and resistance exercises to improve endurance and prevent muscle wasting

When to stop and notify the PT: Symptomatic hypotension or marked dizziness (especially post-dialysis) or a sudden change in mental status are reasons to stop the current activity and notify the supervising PT - not signs to work through.

Example: dizziness after dialysis

A patient with end-stage renal disease arrives for gait training two hours after a hemodialysis session. After a few minutes of walking, the patient reports feeling lightheaded and dizzy. What should the PTA do?

Answer: Stop the activity, have the patient sit or lie down, and check blood pressure on the arm without the AV fistula. Post-dialysis hypotension from rapid fluid removal is the likely cause, which is why exercise is scheduled at least 4 hours after treatment or on non-dialysis days. Report the finding to the supervising PT, who decides whether the session continues.

Urinary Incontinence Overview

  • Involuntary urine loss from bladder, urethra, or pelvic floor dysfunction
  • Four main types distinguished by cause and first-line PT management

Stress & Urge Incontinence

  • Stress: weak pelvic floor/sphincter, leakage with coughing/sneezing/jumping; treat with Kegels (lift-and-squeeze, avoid substituting glutes/hips/abs), bladder diary, biofeedback, pessary, or sling surgery
  • Urge: detrusor overactivity, sudden strong urge (neuro conditions, infection); treat with bladder retraining/scheduled voiding, reduce bladder irritants (caffeine/alcohol), anticholinergics/beta-3 agonists, tibial nerve stimulation

Overflow & Functional Incontinence

  • Overflow: incomplete emptying from obstruction (BPH, prolapse) or weak detrusor (diabetes, SCI); dribbling/weak stream; treat with double/timed voiding, catheterization — pelvic floor strengthening does NOT help (use relaxation training/biofeedback if pelvic floor fails to relax)
  • Functional: mobility/cognitive barriers prevent timely toileting despite normal bladder; treat with environmental modifications, scheduled toileting, mobility aids, caregiver education
  • SCI at T6 or above: distended bladder/blocked catheter can trigger autonomic dysreflexia (hypertension, headache, flushing) — sit patient up, check catheter, notify PT immediately

Pelvic floor function and precautions

  • Correct contraction: squeeze and lift (perineum moves in and up); “the knack” = contract just before a cough, sneeze, or lift
  • Normal voiding about 6-8 times/day, every 3-4 hours
  • Report bearing down instead of lifting, signs of an overactive pelvic floor (pain, frequency/urgency), and fluid restriction (irritates bladder, raises UTI risk, worsens constipation)
  • High-impact or heavy-lift exercise can provoke leakage; diuretics cause frequency/urgency - plan bathroom access

Acute Kidney Injury (AKI)

  • Sudden, often reversible kidney dysfunction; causes: pre-renal (hypovolemia/shock), intra-renal (nephrotoxins/necrosis), post-renal (obstruction/stones)
  • Kidney stones: flank pain radiating to groin/genitals, hematuria — report to PT, don’t treat as musculoskeletal
  • Signs: oliguria, fluid retention, elevated potassium/BUN/creatinine
  • PT: monitor mental status/electrolytes, avoid overexertion (hypotension risk)

Chronic Kidney Disease (CKD)

  • Progressive decline, leading causes: diabetes, hypertension, glomerulonephritis, polycystic kidney disease
  • Staged by GFR: Stage 1 (≥90, kidney damage present) through Stage 5/ESRD (<15 or on dialysis, requires dialysis/transplant)
  • Normal labs: creatinine 0.6-1.2 mg/dL, BUN 7-20 mg/dL, potassium 3.5-5.0 mEq/L
  • Signs: fatigue, edema, anemia, metabolic acidosis, pruritus, uremia (confusion, neuropathy)
  • PT: low-to-moderate exercise, monitor BP/electrolytes, avoid dehydration, watch for bone fracture risk

Nephrotic Syndrome

  • Glomerular damage causes severe proteinuria (>3.5g/day), hypoalbuminemia, hyperlipidemia
  • Severe edema (legs), increased thrombosis/infection risk
  • PT: monitor edema-related mobility limits, caution with clotting risk during exercise

Hemodialysis Considerations

  • Typically 3x/week, 3-5 hrs/session; filters blood in ESRD
  • Complications: post-dialysis hypotension, fatigue, dizziness, cramping, AV fistula/graft issues
  • PT: schedule exercise on non-dialysis days or ≥4 hours post-treatment; never take BP on fistula/graft arm
  • Stop activity and notify PT for symptomatic hypotension, marked dizziness, or sudden mental status change

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Urinary incontinence and renal disorders

Incontinence

Urinary incontinence is the involuntary loss of urine due to dysfunction in the bladder, urethra, or pelvic floor muscles. There are several types, each with different causes and treatments; urodynamic studies evaluate bladder function to classify the type.

Stress incontinence

Cause

  • Weak pelvic floor muscles or urethral sphincter dysfunction
  • Common in postpartum women, postmenopausal women, and after prostate surgery in men
  • Triggered by increased intra-abdominal pressure (e.g., coughing, sneezing, laughing, jumping)

Treatment

  • Pelvic floor muscle training (Kegels) to strengthen muscles: cue a lift-and-squeeze contraction around the urethra and anus without substituting gluteal, hip-adductor, or abdominal muscles. Training progresses from brief, quick contractions (useful for urge suppression) to longer-held endurance contractions, and a bladder diary (tracking fluid intake, voiding times, and leakage episodes) helps guide the plan.
  • Bladder training to improve control
  • Biofeedback & electrical stimulation for muscle activation
  • Pessary (vaginal) or urethral inserts (in some cases to support and hold pelvic organs in place)
  • Surgical options (e.g., sling procedures) for severe cases

Urge incontinence

Cause

  • Detrusor muscle overactivity leads to sudden, strong urges to urinate
  • Can be related to neurological conditions (e.g., stroke, Parkinson’s, MS), infections, or idiopathic causes
    • In older adults a common infection, such as a urinary tract infection, pneumonia or influenza, can cause delirium, which begins suddenly, whereas dementia typically begins gradually

Treatment

  • Bladder retraining (scheduled voiding, delaying urination)
  • Pelvic floor exercises to control urgency
  • Behavioral modifications (reducing caffeine, alcohol, and bladder irritants)
  • Medications (anticholinergics, beta-3 agonists) to relax the bladder
  • Neuromodulation (e.g., tibial nerve stimulation) in severe cases

Overflow incontinence

Cause

  • Incomplete bladder emptying due to obstruction (e.g., enlarged prostate, pelvic organ prolapse) or weak detrusor muscle (e.g., diabetes, spinal cord injury, multiple sclerosis)
  • Symptoms include dribbling, a weak stream, and a feeling of incomplete emptying
  • In spinal cord injury at T6 or above, a distended bladder or a blocked catheter is the most common trigger of autonomic dysreflexia (sudden hypertension, pounding headache, flushing above the lesion): sit the patient up, check the catheter, and notify the PT immediately

Treatment

  • Double voiding technique (waiting and trying to urinate again)
  • Timed voiding to prevent overflow
  • Pelvic floor strengthening does not treat overflow incontinence, because it neither relieves an obstruction nor strengthens a weak detrusor; if a pelvic floor that fails to relax is impeding emptying, pelvic floor relaxation training (often with biofeedback) is used instead
  • Catheterization (intermittent or long-term) for severe cases
  • Surgical intervention if caused by obstruction (e.g., prostate surgery)

Functional incontinence

Cause

  • Physical, structural, or cognitive impairments (e.g., stroke, dementia, arthritis, Parkinson’s, steps to bathroom, physical obstacles) prevent timely access to the bathroom
  • No direct bladder dysfunction, but mobility limitations or mental impairments lead to accidents

Treatment

  • Environmental modifications (accessible bathrooms, commodes, grab bars)
  • Scheduled toileting based on routine
  • Mobility training & assistive devices
  • Caregiver education for dementia-related cases

The following table compares the four types of urinary incontinence by cause, presentation, and first-line physical therapy management.


Type Cause Presentation First-line PT management
Stress Weak pelvic floor muscles or urethral sphincter Leakage with coughing, sneezing, laughing, or jumping Pelvic floor muscle training (Kegels), biofeedback
Urge Detrusor overactivity Sudden, strong urge; leakage before reaching the toilet Bladder retraining (scheduled voiding, delaying urination), behavioral modification
Overflow Obstruction or a weak detrusor, so the bladder does not empty Dribbling, weak stream, feeling of incomplete emptying Double and timed voiding, catheterization when needed; not pelvic floor strengthening
Functional Mobility or cognitive barriers; the bladder itself works Accidents because the toilet cannot be reached in time Environmental modification, scheduled toileting, mobility training

Pelvic floor function and exercise precautions

A correct pelvic floor contraction squeezes around the pelvic openings and lifts the pelvic floor; the perineum moves inward and up, not outward. Two everyday uses follow from that:

  • The knack: contracting the pelvic floor just before a cough, sneeze, or lift, so it is already braced when abdominal pressure rises.
  • Normal voiding: about 6-8 voids a day, every 3-4 hours. A bladder diary is read against this interval, and timed voiding works toward it.
  • Nocturia: the need to get up regularly at night to urinate; about 50% of adults older than 65 get up at least once a night, and about a quarter have two or more episodes.

Bladder and pelvic floor programs have their own adverse effects to watch for and report to the PT:

  • Bearing down instead of lifting: straining (pushing downward, often with breath-holding) is a common error that raises intra-abdominal pressure and loads the pelvic floor - the opposite of the exercise’s goal.
  • Overactive pelvic floor: a pelvic floor that can’t fully relax is linked to pelvic pain, urinary frequency and urgency, and difficulty with bowel movements. Strengthening isn’t always appropriate here; relaxation training is used instead.
  • Fluid restriction: patients often drink less to leak less, but concentrated urine irritates the bladder and promotes urgency, frequency, and urinary tract infections, and restricting fluids worsens constipation.

Interventions for other problems can also provoke leakage:

  • High-impact and heavy-lift exercise: running, jumping, landing, and heavy lifting raise intra-abdominal pressure, and incontinence is common in high-impact sports and in weightlifters. Report new leakage during exercise to the PT.
  • Diuretics: increase urine production, causing frequency and urgency, and can lead to urge leakage in an older patient who moves slowly. Plan bathroom access, and where possible schedule sessions away from the hours right after the dose.

Renal disorders

Common renal disorders

The kidneys play a vital role in filtering waste, regulating electrolytes, and maintaining fluid balance. Dysfunction can lead to serious systemic issues, including metabolic imbalances and cardiovascular complications.

Acute kidney injury (AKI)

Definition: Sudden loss of kidney function due to ischemia, toxins, or trauma. It is often reversible if treated early.

Causes:

  • Pre-renal: Hypovolemia, shock, heart failure.
  • Intra-renal: Acute tubular necrosis, nephrotoxic drugs, infections
  • Post-renal: Obstruction (kidney stones, tumors, BPH). Kidney stones (renal calculi) typically present with sudden-onset flank pain and hematuria; as the stone travels down the ureter, pain can radiate to the lower abdomen, groin, and genital region. This radiating pain can mimic musculoskeletal pain, so report it to the supervising PT rather than treating it as a musculoskeletal complaint.

Signs and symptoms:

  • Oliguria (low urine output) and fluid retention
  • Electrolyte imbalance (increased potassium) and abnormal labs (increased BUN and creatinine)

PT considerations:

  • Monitor for fatigue, altered mental status, and electrolyte imbalances
  • Avoid overexertion due to risk of hypotension and electrolyte shifts

Chronic kidney disease (CKD)

Definition: Progressive decline in kidney function, leading to end-stage renal disease (ESRD) if untreated

Causes:

  • Diabetes (leading cause), hypertension, glomerulonephritis, polycystic kidney disease

The following table lists the five stages of chronic kidney disease by glomerular filtration rate (GFR) and clinical picture.


Stage GFR (mL/min/1.73 m²) Clinical picture
1 ≥90 (with kidney damage) Mild damage, no major symptoms
2 60-89 Mild damage, no major symptoms
3 30-59 Moderate impairment; possible anemia, bone mineral disease
4 15-29 Severe dysfunction; dialysis preparation begins
5 (ESRD) <15, or on dialysis Requires dialysis or a kidney transplant

Approximate normal reference ranges are serum creatinine 0.6-1.2 mg/dL, BUN 7-20 mg/dL, and potassium 3.5-5.0 mEq/L; values above these ranges reflect the waste-product and electrolyte imbalances described below. Imaging: renal ultrasound checks for obstruction, stones, or structural changes.

Signs and symptoms:

  • Fatigue, muscle cramps, edema, anemia, metabolic acidosis, hypertension, pruritus (intense itching, most common in advanced CKD and ESRD)
  • Uremia (buildup of waste in blood) → nausea, confusion, neuropathy

PT considerations:

  • Monitor for fatigue, blood pressure changes, and electrolyte imbalances
  • Encourage low-to-moderate intensity exercise to improve functional capacity
  • Avoid excessive fluid loss (dehydration can worsen kidney function)
    • Dehydration causes dry mouth and reduced urine production, and in severe cases blood pressure falls, causing light-headedness or fainting, particularly upon standing
  • Be aware of bone mineral disease & risk of fractures

Nephrotic syndrome

Definition: A kidney disorder causing severe protein loss in urine due to damage to the glomeruli

Signs and symptoms:

  • Severe edema (especially in the legs), proteinuria (>3.5g/day), hypoalbuminemia, hyperlipidemia.
  • Increased risk of thrombosis and infections

PT considerations:

  • Monitor for edema-related mobility restrictions
  • Be cautious of increased clotting risk during exercise

Hemodialysis (HD) considerations

Hemodialysis is used in ESRD to filter blood through a machine when the kidneys can no longer perform their function. It is typically done 3 times per week for 3-5 hours per session.

Common complications:

  • Hypotension during or after dialysis (due to rapid fluid removal)
  • Fatigue, dizziness, cramping (electrolyte shifts)
  • Access site issues (fistula/graft infections, thrombosis)

PT considerations:

  • Schedule exercise on non-dialysis days or at least 4 hours after treatment; avoid exercise immediately before or after dialysis because of fatigue and hypotension risk
  • Do not take blood pressure on the arm with an AV fistula/graft to prevent complications
  • Encourage low-impact aerobic and resistance exercises to improve endurance and prevent muscle wasting

When to stop and notify the PT: Symptomatic hypotension or marked dizziness (especially post-dialysis) or a sudden change in mental status are reasons to stop the current activity and notify the supervising PT - not signs to work through.

Example: dizziness after dialysis

A patient with end-stage renal disease arrives for gait training two hours after a hemodialysis session. After a few minutes of walking, the patient reports feeling lightheaded and dizzy. What should the PTA do?

Answer: Stop the activity, have the patient sit or lie down, and check blood pressure on the arm without the AV fistula. Post-dialysis hypotension from rapid fluid removal is the likely cause, which is why exercise is scheduled at least 4 hours after treatment or on non-dialysis days. Report the finding to the supervising PT, who decides whether the session continues.

Key points

Urinary Incontinence Overview

  • Involuntary urine loss from bladder, urethra, or pelvic floor dysfunction
  • Four main types distinguished by cause and first-line PT management

Stress & Urge Incontinence

  • Stress: weak pelvic floor/sphincter, leakage with coughing/sneezing/jumping; treat with Kegels (lift-and-squeeze, avoid substituting glutes/hips/abs), bladder diary, biofeedback, pessary, or sling surgery
  • Urge: detrusor overactivity, sudden strong urge (neuro conditions, infection); treat with bladder retraining/scheduled voiding, reduce bladder irritants (caffeine/alcohol), anticholinergics/beta-3 agonists, tibial nerve stimulation

Overflow & Functional Incontinence

  • Overflow: incomplete emptying from obstruction (BPH, prolapse) or weak detrusor (diabetes, SCI); dribbling/weak stream; treat with double/timed voiding, catheterization — pelvic floor strengthening does NOT help (use relaxation training/biofeedback if pelvic floor fails to relax)
  • Functional: mobility/cognitive barriers prevent timely toileting despite normal bladder; treat with environmental modifications, scheduled toileting, mobility aids, caregiver education
  • SCI at T6 or above: distended bladder/blocked catheter can trigger autonomic dysreflexia (hypertension, headache, flushing) — sit patient up, check catheter, notify PT immediately

Pelvic floor function and precautions

  • Correct contraction: squeeze and lift (perineum moves in and up); “the knack” = contract just before a cough, sneeze, or lift
  • Normal voiding about 6-8 times/day, every 3-4 hours
  • Report bearing down instead of lifting, signs of an overactive pelvic floor (pain, frequency/urgency), and fluid restriction (irritates bladder, raises UTI risk, worsens constipation)
  • High-impact or heavy-lift exercise can provoke leakage; diuretics cause frequency/urgency - plan bathroom access

Acute Kidney Injury (AKI)

  • Sudden, often reversible kidney dysfunction; causes: pre-renal (hypovolemia/shock), intra-renal (nephrotoxins/necrosis), post-renal (obstruction/stones)
  • Kidney stones: flank pain radiating to groin/genitals, hematuria — report to PT, don’t treat as musculoskeletal
  • Signs: oliguria, fluid retention, elevated potassium/BUN/creatinine
  • PT: monitor mental status/electrolytes, avoid overexertion (hypotension risk)

Chronic Kidney Disease (CKD)

  • Progressive decline, leading causes: diabetes, hypertension, glomerulonephritis, polycystic kidney disease
  • Staged by GFR: Stage 1 (≥90, kidney damage present) through Stage 5/ESRD (<15 or on dialysis, requires dialysis/transplant)
  • Normal labs: creatinine 0.6-1.2 mg/dL, BUN 7-20 mg/dL, potassium 3.5-5.0 mEq/L
  • Signs: fatigue, edema, anemia, metabolic acidosis, pruritus, uremia (confusion, neuropathy)
  • PT: low-to-moderate exercise, monitor BP/electrolytes, avoid dehydration, watch for bone fracture risk

Nephrotic Syndrome

  • Glomerular damage causes severe proteinuria (>3.5g/day), hypoalbuminemia, hyperlipidemia
  • Severe edema (legs), increased thrombosis/infection risk
  • PT: monitor edema-related mobility limits, caution with clotting risk during exercise

Hemodialysis Considerations

  • Typically 3x/week, 3-5 hrs/session; filters blood in ESRD
  • Complications: post-dialysis hypotension, fatigue, dizziness, cramping, AV fistula/graft issues
  • PT: schedule exercise on non-dialysis days or ≥4 hours post-treatment; never take BP on fistula/graft arm
  • Stop activity and notify PT for symptomatic hypotension, marked dizziness, or sudden mental status change

More from Other systems

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