Cardiac exercise testing
Types of test
- 6 minute walk test
- Test goal: measure distance walked in 6 minutes with or without assistive device
- Rationale: test for endurance related to cardiovascular health
- Setting used: Phase I and Phase II of cardiac rehabilitation
- Test advantages: Quick assessment of endurance that can be completed for lower level patients
- Graded exercise testing
- Test goal: assess the maximal workload that can be applied to the cardiovascular system via treadmill testing at different grades (incline)
- Rationale: provide therapist with information on highest capacity of exercise tolerance
- Provides maximal heart rate that can be used to then give target heart range with activity
- Setting: Phase II cardiac rehabilitation
- Test advantages: Most accurate assessment tool of an individual’s exercise tolerance
- Cycler ergometer test:
- Test goal: submaximal test utilized to assess the VO2 max through graded exercise (increasing in resistance)
- Rational: provide VO2 max information that can then be placed in equation to calculate maximal heart rate
- Setting: Phase II cardiac rehabilitation
- Test advantage: Utilized over maximal exercise testing if patient unable to tolerate maximal testing parameters
Contraindications for exercise testing
- Unstable angina
- Acute myocardial infarction
- Acute congestive heart failure
- Uncontrolled arrhythmias
- Systolic blood pressure >200 mmHg or diastolic blood pressure >115 mmHg
- Severe anemia
- Hemoglobin less than 8
- Advanced or complicated pregnancy
Reasons to stop exercise testing
- Severe ST segment depression of 2mm or greater
- Cardiac arrest
- Development of new 2nd or 3rd degree heart block
- Patient requests to stop
- Severe chest pain, dizziness, or dyspnea
- Fall in systolic blood pressure <20 mmHg
- Ataxia
- Rise in systolic blood pressure >200 mmHg or diastolic > 110 mm Hg
Cardiac medications
- Beta blockers (atenolol): hypertension medication
- Mechanism of action: works on the heart muscle by blocking epinephrine and causing vasodilators
- PT implications: do not use heart rate as indicator for response to exercise; use perceived exertion scale (RPE) to assist with tolerance to activity
- Calcium channel blockers: hypertension medication
- Mechanism of action: works on heart muscle to prevent calcium from entering heart tissue; decreasing contractility
- PT implications: orthostatic hypotension
- Angiotensin converting enzyme inhibitors (ACE Inhibitors): hypertension medications
- Mechanism of action: works on kidneys to stop the conversion of Angiotensin I to converting to Angiotensin II and causes vasodilation in peripheral vascular system
- PT implications: Orthostatic hypotension, dizziness, palpitations
- Diuretics (lasix & hydrochlorothiazide): hypertension medication
- Mechanism of action: works on kidneys to decrease the fluid retention in body by moving sodium out of the body
- PT implications: electrolyte imbalance, orthostatic hypotension, muscle cramps,
increased urination
- Anticoagulants (warfarin, coumadin): decreases clotting of blood
- Mechanism of action: works to reduce the clotting within blood
- PT implications: hemorrhage risk, can easily bruise
- Nitroglycerin: used specifically for angina pectoralis
- Mechanism of action: work to relax smooth muscle in the heart by release of nitric oxide which will cause vasodilation and reduction in blood pressure
- Specifically used in angina pectoris
- PT implications: dizziness, headache, weakness, nausea
- Mechanism of action: work to relax smooth muscle in the heart by release of nitric oxide which will cause vasodilation and reduction in blood pressure
- Digitalis (digoxin): congestive heart failure medication
- Mechanism of action: inhibits AV node activation
- PT implications: decreased renal failure, increased risk for toxicity (digoxin toxicity) which can lead to hospitalization
- Antihyperlipidemias (statins): cholesterol mediation
- Mechanism of action: blocks production of cholesterol by liver
- PT implications: muscle pain, muscle weakness, fatigue, diarrhea, constipation, headache
- Amiodorone (antiarrhythmic agent): unstable ventricular tachycardia and recurrent ventricular fibrillation medication
- Mechanism of action: increases the duration of the action potential as well as the effective refractory period for cardiac cells
- PT implications: signs of congestive heart failure, including dyspnea, rales/crackles, peripheral edema, jugular venous distention, and exercise intolerance
Observational cardiac topics
Pulse (also known as heart beat or rhythm)
- 0= absent
- 1+ = weak/thready
- 2+= normal
- 3+= increased
- 4+= bounding
Pitting edema grading (associated with right sided heart failure):
- Grade 1: 0-2 mm indentation and rebounds immediately
- Grade 2: 3-4 mm indentation and rebounds within 15 seconds
- Grade 3: 5-6 mm indentation and rebounds within 30-60 seconds
- Grade 4: 8mm or more indentation and rebounds 60 seconds or longer
Auscultation of heart sounds
Image #7

- S1:
- Normal heart sound
- First heart sound when atrioventricular (mitral and tricuspid) valve close
- Heard at the 5th intercostal space at midclavicular line
- Signals the beginning of systole
- Often referred to as “lub”
- S2
- Normal heart sound
- Second heart sound when semilunar (aortic and pulmonary) valves close
- Heart at at the second intercostal space along the right sternal boarder
- Signals the beginning of diastole
- Often referred to as “dub”
- S3
- Adventitious heart sound in adults and normal in children
- Heard between 4th and 5th ribs on left side of chest
- Associated with rapid filling of ventricles during early diastole
- Often associated with fluid overload or weakened left ventricle which can be indicative heart failure, myocardial infarction, dilated cardiomyopathy, valvular regurgitation, or constrictive pericarditis
- S4
- Adventitious heart sound in adults and children
- Heard between 4th and 5th ribs on left or right side of chest
- Associated with reduced ventricular compliance or increased resistance to ventricular filling
- Often associated with left ventricular hypertrophy or left sided heart failure